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Amyloid Beta (25-35) Peptide (Y-0044)  
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1mg/400.00元
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產(chǎn)品編號(hào) Y-0044
英文名稱 Amyloid Beta (25-35) Peptide
中文名稱 β淀粉樣肽25-35
別    名 Amyloid beta Peptide (25-35); Beta-Amyloid (25-35); Amyloid beta-Protein Fragment 25-35; A beta 25-35; Beta Amyloid 25-35.  Aβ淀粉樣肽25-35; β淀粉樣肽(25-35); Amyloid β (25-35); Aβ 25-35; β Amyloid (25-35); β Amyloid 25-35.
Specific References  (7)     |     Y-0044 has been referenced in 7 publications.
[IF=6.057] Mingxing Liu. et al. A sensitive ratiometric biosensor for determination cardiac troponin I of myocardial infarction markers based on N, Zn-GQDs. TALANTA. 2022 May;:123577  
[IF=6.057] Limin Zhou. et al. Copper doped terbium metal organic framework as emitter for sensitive electrochemiluminescence detection of CYFRA 21-1. Talanta. 2022 Feb;238:123047  
[IF=2.89] Zhang, Gui-lian, et al. Edaravone Ameliorates Oxidative Damage Associated with Aβ25-35 Treatment in PC12 Cells. Journal of Molecular Neuroscience (2013): 1-10.2.  Other ;  
[IF=2.883] Jiang Z et al. N-Acetyl chitooligosaccharides attenuate amyloid β-induced damage in animal and cell models of Alzheimer's disease. Process Biochemistry.2019  Other ;  
[IF=2.1] Zhang, Beiru, et al. "The Toxicity Mechanisms of Action of Aβ25–35 in Isolated Rat Cardiac Myocytes." Molecules 19.8 (2014): 12242-12257.  Other ;  Rat.  
[IF=2.08] Teng, Yan, et al. ?Compound danshen tablet ameliorated abeta25-35-induced spatial memory impairment in mice via rescuing imbalance between cytokines and neurotrophins.? BMC Complementary and Alternative Medicine 14.1 (2014): 23.  Immunogen ;  Mouse.  
[IF=1.41] Ji S et al. Protective role of phenylethanoid glycosides, Torenoside B and Savatiside A, in Alzheimer's disease.Exp Ther Med. 2019 May;17(5):3755-3767.  Other ;  
理論分子量 1.06kDa
性    狀 Lyophilized
序    列 GSNKGAIIGLM
純化方法 HPLC
活性 Yes
保存條件 Shipped at 4℃. Stored at -20℃ for one year. Avoid repeated freeze/thaw cycles.
產(chǎn)品介紹
Aβ是老年性癡呆發(fā)病的始動(dòng)因子,并證實(shí)引起毒性主要部位在第25-35氨基酸之間(Aβ25-35) ,Aβ25-35(Aβ25-35 peptide)具有神經(jīng)毒性,可使海馬和隔區(qū)神經(jīng)元的存活率下降,繼而發(fā)生神經(jīng)元凋亡的形態(tài)學(xué)改變.
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